AP Psychology — Interactions of Heredity and Environment

Unit: Biological Bases of Behavior • Focus: Behavioral genetics, heritability, twin/adoption designs, epigenetics, and gene–environment interaction.

Key Terms — Click to Expand

Your genetic makeup — the set of genes you inherit.
Observable traits or behaviors — how genes are expressed under environmental influence.
The proportion of variation in a population’s trait that can be attributed to genetic differences.
Study of how genes and environment jointly shape behavior using twin and adoption studies.
MZ vs DZ comparisons estimate genetic contribution; adoption studies separate environment from heredity.
Study of how external factors switch genes on/off without altering DNA sequence (e.g., methylation).
Genes set upper/lower limits; environment determines where within that range a trait manifests.

Concepts to Understand — Click to Expand

Genes provide instructions, but environment and experience affect expression and outcome.
Heritability applies to population variation, not individuals—high heritability ≠ unchangeable trait.

Definition: rGE stands for gene–environment correlation—how an individual’s genetic makeup influences the environments they experience.

Different from G×E: rGE means genes shape exposure to environments, while G×E means the effects of genes depend on the environment.

Three types of rGE:

  • Passive: Parents provide both genes and matching environments.
    Example: Musical parents pass on musical ability and fill the home with instruments.
  • Evocative: A person’s traits evoke reactions from others.
    Example: A sociable child elicits more attention and praise.
  • Active: People seek out settings that fit their tendencies.
    Example: A risk-taking teen joins extreme-sports peers.

Key idea: rGE creates feedback loops—genes influence behavior, which shapes environments that further reinforce those genetic effects.

Gene–Environment Correlation Diagram

Diagram illustrating rGE feedback loop: Genes → Behavior → Environment → Reinforcement.
Image: Wikimedia Commons, CC BY-SA.

Big Five Personality Self-Assessment (approx. 8 minutes)

Students may take this free, research-aligned Big Five test to discover their traits (Openness, Conscientiousness, Extraversion, Agreeableness, Neuroticism):

➡ Take the IPIP-50 Big Five Inventory

Warm-Up: “Who Are You — Nature or Nurture?” (approx. 10 minutes)

Write 3–4 sentences: pick one personality trait and estimate what percentage you think comes from your genes vs your environment. Then pair-share and discuss: How could scientists test this?

Mini-Lecture: Nature, Nurture, and Interaction (approx. 25 minutes)

  • Genes → DNA → chromosomes → genome → genotype vs phenotype
  • Twin/adoption research and behavioral genetics
  • Heritability and environmental influence
  • Epigenetics: methylation, histones, gene regulation
  • Gene–environment interaction and plasticity

Activity: Twin & Adoption Data Simulation (approx. 20 minutes)

Group Instructions:

  1. Analyze correlations for IQ, extraversion, and depression across groups.
  2. Rank each trait from most genetic → most environmental.
  3. Prepare one-slide/poster with your findings and example.
Pair TypeIQExtraversionDepression
MZ twins (together)0.860.540.44
MZ twins (apart)0.750.420.34
DZ twins (together)0.580.280.18
Adoptive siblings0.200.080.05

Big Five: Your Guess vs What Research Finds

Student prompt: On a sticky note (or quick poll), guess your own mix of genes vs environment for each Big Five trait (O, C, E, A, N). Then open the dropdowns below to compare with typical research estimates and examples.

Reminder: These are population-level estimates (heritability ≠ immutability). Most modern studies find both genes and environment matter, with nonshared experiences often outweighing shared home effects.

Across many twin/adoption studies, Big Five traits commonly show moderate heritability (~40–60%). The remainder reflects environment, especially nonshared (unique) experiences. A simple way to remember:

TraitGenetic (heritability)Environment
Openness~45–60%~40–55% (mostly nonshared)
Conscientiousness~40–50%~50–60% (mostly nonshared)
Extraversion~45–55%~45–55% (mostly nonshared)
Agreeableness~40–50%~50–60% (mostly nonshared)
Neuroticism~40–50%~50–60% (mostly nonshared)

Teacher note: Ranges vary by sample, age, and methods; use these as ballpark figures to anchor discussion. High heritability does not mean “unchangeable.”

Typical genetic influence: ~45–60%. Environment: ~40–55%, largely nonshared.

  • Hereditary/biological contributors (examples): Temperamental curiosity; neural networks supporting cognitive exploration and novelty processing.
  • Environmental contributors: Enrichment/exposure (books, travel, arts/STEM clubs), teaching styles encouraging inquiry, diverse peer groups, cultural norms about creativity.
  • rGE examples: Curious students may seek enrichment (active rGE); curious parents may provide stimulating homes (passive rGE).
  • G×E idea: Enriched classrooms may amplify openness in students already inclined toward exploration.

Typical genetic influence: ~40–50%. Environment: ~50–60%, largely nonshared.

  • Hereditary/biological contributors: Temperamental effortful control, self-regulation capacities, reward sensitivity for long-term goals.
  • Environmental contributors: Family routines, study-skill coaching, academic expectations, sports/work responsibilities, feedback systems, sleep and health habits.
  • rGE: Conscientious teens may choose structured activities that further train organization (active rGE).
  • G×E: Skills programs (planners, checklists) may yield bigger gains for students with certain self-regulation propensities.

Typical genetic influence: ~45–55%. Environment: ~45–55%, largely nonshared.

  • Hereditary/biological contributors: Baseline sociability/approach tendencies; sensitivity to social reward and positive affect.
  • Environmental contributors: Peer networks, opportunities for leadership/performance, classroom climate for participation, family modeling of sociability, cultural norms.
  • rGE: Sociable students evoke invitations/attention (evocative rGE) and join social clubs (active rGE).
  • G×E: Supportive, low-threat contexts can enhance social engagement in those already dispositionally outgoing.

Typical genetic influence: ~40–50%. Environment: ~50–60%, largely nonshared.

  • Hereditary/biological contributors: Temperamental warmth/empathy, threat reactivity and prosocial motivation systems.
  • Environmental contributors: Parenting style (warmth/induction), conflict-resolution teaching, cooperative classrooms/teams, cultural expectations around politeness/helping.
  • rGE: Warm, prosocial kids receive more positive peer responses (evocative rGE), reinforcing agreeableness.
  • G×E: SEL programs may especially benefit youth with latent prosocial tendencies.

Typical genetic influence: ~40–50%. Environment: ~50–60%, largely nonshared.

  • Hereditary/biological contributors: Temperamental negative affectivity, stress reactivity/sensitivity.
  • Environmental contributors: Chronic stress, family conflict, peer victimization, sleep deprivation, coping skills training, therapy/CBT exposure, exercise.
  • rGE: Highly reactive individuals may encounter (or perceive) more stressors, shaping environments that maintain anxiety/rumination.
  • G×E: Stress-reducing interventions can have outsized effects for those with higher biological sensitivity to stress.

Teacher prompt: Ask: “Which of your guesses matched these patterns? Where did your life experiences push you away from your genetic ‘starting point’?”

Video Segment (approx. 10 minutes)

Crash Course Biology: Nature? Nurture? Not So Simple

Epigenetics Explained — Penn Medicine

Molecular Biology Animations (approx. 12 minutes)

DNA Packaging (Nucleosomes) — WEHI

Histone Acetylation — Khan Academy

Interactive: Gene Control — Add/Remove Epigenetic Tags

Interactive Discussion (approx. 20 minutes)

  • Law 1: All human behavioral traits are heritable (to some degree).
  • Law 2: The effect of being raised in the same family is smaller than the effect of genes.
  • Law 3: A substantial portion of the variation in complex human behavioral traits is not accounted for by genes or families (nonshared environment).
  • “Which law surprises you most and why?”
  • “Give an example of a nonshared environmental influence.”
  • “How could the same home produce different outcomes for siblings?”
  • Myth: “Heritable” = “unchangeable.”
    Fix: Emphasize population statistic; environment and interventions still matter.
  • Myth: Shared family environment is irrelevant.
    Fix: It matters, but often less than genes and nonshared experiences for many traits.

High heritability means within this population, given these environments, genes explain more of the variation—not that the trait can’t change.

  • “Height has high heritability, yet nutrition changes population height over time—how?”
  • “If two schools differ in resources, how might that alter the heritability of achievement?”

1-minute write: Explain to a family member why a highly heritable trait can still respond to environmental change.

Genes define a potential range; environment determines where outcomes fall within that range.

  • Show photos (or real plants) grown under rich vs poor soil/light.
  • Ask: “If genes are identical, why do heights differ?”
  • Connect to academic opportunity and health environments.
  • Pitfall: Assuming the range is the same for everyone.
    Extension: Discuss polygenic scores and different ranges.
  • Extension: Have students propose human “soil/light” analogies (nutrition, schooling, stress).

Small-Group Scenarios (approx. 15 minutes)

Task: Identify genetic factors, environmental differences, and how they interact; propose a simple study.

  • High MZ similarity suggests genetic influence; differences highlight environmental effects.
  • Discuss shared prenatal vs postnatal environments.

Correlate twin scores on trait X; compare means across SES contexts; control for age/sex; discuss limitations (nonrandom separation).

Task: Map diathesis–stress: genetic predisposition × home stress.

  • Prompt: “What protective factors buffer risk?” (warm parenting, CBT skills, sleep)
  • Myth: Genes = fate → Reframe as sensitivity to context.

Use anxiety scales + home stress index; test interaction term (stress × genotype/proxy).

Task: Is this rGE (musical family) or G×E (lessons amplify disposition)? Likely both.

  • Distinguish passive rGE (musical home) from active rGE (student joins band).
  • G×E: Lessons (E) strengthen expression of aptitude (G).

Task: Describe how methylation patterns may alter gene expression and potentially influence descendants.

  • Prompt: “What mechanisms could transmit effects across generations?”
  • Caution: Human causal claims are hard—observational limits; triangulate with animal models.

Check for Understanding (approx. 10 minutes)

Expected idea: The proportion of variation in a trait within a population attributable to genetic differences.

Scoring tip: Include “variation” + “genetic differences” for full credit.

Expected idea: Genotype = genetic makeup; Phenotype = expressed traits resulting from interaction with environment.

Misconception alert: Don’t say phenotype is “just genes.” Emphasize environmental influence.

Expected idea: Genes set upper and lower limits; environment determines the actual outcome within that range.

Extension: Provide an example (e.g. nutrition and height).

Expected idea: Environmental factor (e.g. stress, diet, toxin) that changes DNA methylation or histone state, altering gene expression.

Tip: Name both the environmental factor and its molecular mechanism or effect.

Expected idea: Genes and environments correlate via passive, evocative, or active processes (e.g. a sociable child elicits more social interactions).

Prompt: Ask which rGE type (passive, evocative, active) fits their example.

Closure & Homework (approx. 10 minutes)

Homework: Watch Moshe Szyf’s TED Talk “How Early Life Experience Is Written into DNA” and write one paragraph connecting it to epigenetics.

Teaching Images (Embedded — No External Links)

DNA Methylation at CpG Site C G CH₃ DNA Methylation (CpG) Cytosine Guanine Methyl group Often reduces transcription by tightening chromatin or blocking TF binding
DNA methylation at a CpG site: a methyl group (CH₃) added to cytosine tends to reduce transcription.
Histone Acetylation & Chromatin Loosening Tight chromatin (low transcription) Loose chromatin (higher transcription) Ac Ac histone acetylation
Histone acetylation (Ac) relaxes chromatin by neutralizing histone tail charge → greater access for transcription.
Nucleosome & Chromatin Packaging Histone core Next nucleosome DNA packaging into nucleosomes DNA wraps ~1.7 turns around histone octamer Linker DNA
DNA wraps around histone cores to form nucleosomes; linker DNA connects nucleosomes to build chromatin.

These are vector diagrams embedded directly in the page, so they should display everywhere (even if external images are blocked).